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Abstract Details

Anti-GAD Antibody Associated Encephalitis: A Child with Epilepsia Partialis Continua and Ataxia
Autoimmune Neurology
P2 - Poster Session 2 (5:30 PM-6:30 PM)
15-020

To review the clinical presentation, laboratory findings, EEG abnormalities, MRI features and treatment response in a child presenting with Epilepsia Partialis Continua (EPC) and Ataxia

Anti-glutamic acid decarboxylase (anti-GAD65) antibodies cause a variety of clinical syndromes. Anti-GAD antibody-associated encephalitis is rare even in adults; only a handful of cases have been described in children, usually with limbic encephalitis and fulminant intractable epilepsy. Many of the pediatric case reports demonstrate significant cognitive impairment with delayed diagnosis. We report an exceptional case in a pediatric patient where early detection and treatment led to rapid and complete resolution of symptoms.
Case Report

The patient was a 10-year-old previously healthy and developmentally normal girl who presented with intermittent left sided facial twitching with preserved awareness, then progressed to continuous facial twitching concerning for EPC. Otherwise, the initial neurological examination was unremarkable.

 

MRI brain with gadolinium revealed foci of cortical and subcortical T2 hyperintensities in the right frontal operculum, right inferior frontal gyrus, the right cingulate gyrus, and the cortex along the left superior frontal sulcus.  EEG showed right frontal and central sharp waves as well as continuous slowing in the right frontal, central and temporal regions. CSF and serum analyses were remarkable for very high anti-GAD65 antibody titers.

 

Anti-seizure medications provided minimal and transient improvement. She subsequently developed ataxia and diplopia. Pulse steroids and therapeutic plasma exchange resulted in complete resolution of EPC, ataxia and diplopia. She was discharged with a plan to continue immunotherapy with rituximab.

This case represents a unique presentation of anti-GAD antibody-associated encephalitis in a pediatric patient with involvement of multiple regions of the brain. Pulse steroids and therapeutic plasma exchange were well tolerated and led to complete resolution of symptoms. Prompt diagnosis and aggressive management is crucial in this likely underdiagnosed and undertreated syndrome in children.
Authors/Disclosures
Isaac Molinero, MD (Ochsner Health)
PRESENTER
Dr. Molinero has nothing to disclose.
Vishal Mandge, MD Dr. Mandge has received personal compensation in the range of $500-$4,999 for serving as a Consultant for EMD Serono.
Susan E. Duberstein Coad, MD Dr. Duberstein Coad has nothing to disclose.