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Abstract Details

Myoclonus – A Rare Presentation of Myxedema Coma
General Neurology
P3 - Poster Session 3 (5:30 PM-6:30 PM)
4-047

To identify an uncommon neurological presentation of myxedema coma

Thyroid hormones are essential for neuronal growth. Hypothyroidism is associated with various neurological complications including impaired cognition, myopathy, nerve entrapment syndromes, sensory deficits, ataxia, cranial neuropathies and, in severe cases, myxedema coma. Myoclonus has not been reported as a presentation of myxedema coma.

Case report

77 year old female with diabetes and hypertension was found unresponsive at her nursing home. Family had noticed a progressive decline in mental status over the past two weeks. Patient appeared lethargic, wandered off during conversation, tried to grab nonexistent objects and her speech was nonsensical occasionally.

 On initial assessment by EMS, patient had bradycardia (42 bpm), temperature 84 F, blood pressure 101/54 and saturation 94%. Patient was obtunded, and noted to have myoclonus of bilateral upper and lower extremities.

 Initial differentials for jerky movements included seizures, focal CNS pathology, hypoxic injury, and metabolic or infectious etiology. CT head and ABG were normal. Patient was loaded with Levetiracetam and continuous video EEG monitoring was started. It demonstrated tri-phasic waves and multifocal myoclonus did not have any electrographic correlation. Detailed encephalopathy workup ordered on admission revealed TSH 126 and low FT4. Endocrinology was consulted and patient started on IV levothyroxine at 100mcg daily and IV hydrocortisone. Patient’s mentation gradually improved. Decrease in frequency and amplitude of myoclonic jerks was noted in response to maintenance therapy with Levetiracetam.

Myxedema coma is characterized by confusion, lethargy or coma in concurrence with hypothermia, hypotension, bradycardia and hypoventilation. Rare but recognized presentations of myxedema coma include focal or generalized seizures, status epilepticus, and psychosis. However, it is important to recognize that myoclonus can also be a presenting symptom of myxedema coma. Secondly, myxedema coma should be considered in the differential when investigating underlying causes of myoclonic activity.

Authors/Disclosures
Ramsha Malik, MD (Willis Knighton Pierremont Neurology clinic)
PRESENTER
No disclosure on file
Allison M. Boyle, MD (UT Houston) No disclosure on file